If vaccines stimulate the body to produce antibodies, but antibodies to Covid-19 break down or disappear over time, does that mean many people will have to keep being revaccinated time and time again?

For context this is a question I answered on Quora

The rapid decline in estimated VE for the modRNA transfections has nothing to do with antibodies breaking down. Antibodies are meant to quickly break down and be a temporary short lived humoral response to an antigen or pathogen during an infection. Immunization is supposed to establish long lived antibody secreting plasma cells, memory B cells and t cells to maintain the immune system’s ability to produce those antibodies and react to future iterations of the pathogen. As I mentioned in a prior answer the modRNA transfections fail to induced detectable and sufficient long lived antibody secreting plasma cells against the spike receptor binding domain. That is why the so called “robust protection” rapidly fades within a few months after the antibody peak at 2–4 weeks post booster. Repeated modRNA transfections not only don’t induce long lived antibody secreting plasma cells against the spike receptor binding domain, that would confer infection risk reduction for more than a few months, they also induce a subclass switch in the anti-receptor binding domain antibodies from IgG1 dominant to IgG4 dominant that down regulates the FC mediated antibody effector functions, induces immune tolerance to the spike protein by blocking other antibody subclasses from stimulating complement immune responses and can lead to antibody dependent enhancement of disease by binding to viral surfaces and incorporating them into white blood cells that become host for viral replication. Unfortunately for recipients that is not the only way the modRNA transfections eventually suppress their immune system. A recent peripheral blood mono-nuclear cell study, published in the Journal of Allergy and Clinical Immunology, discovered that circulating receptor binding domain spike in vaxxed participants triggered a cascade of programmed T cell death through elevated Angiotensin II that induced white blood cells to release free radicals. As I mentioned in a prior post last year, Vitamin D3 indirectly inhibits angiotensin II synthesis through the renin-angiotensin system and also increases Angiotensin-converting enzyme 2 expression.